The problem here seems to be that this doesn't measure how feasible it is to reverse the tendency for the immune system to attack the nerves. Even if you erradicate the causative bacteria, once the immune system has started to attack the nerves, it's possible that it won't stop. Maybe the presence of the antigens (the proteins that are attacket) in the bacteria is needed as the trigger that starts the attack, but once that's gone, the low level presence of the same antigens in the nerves might be enough to sustain the attack. On a more optimistic note, maybe the erradication of the bacteria leads to a lack of simulation that causes the aggressive cells tow ither and die. This must be tested experimentally.
If the autoimmune ttack doesn't stop after erradication of bacteria, maybe a case can be made for aggressive prevention with testing for those bacteria periodically, ,especially in families predisposed to the disease, and prompt erradication once they appear. Maybe there are correlations between diet and the presence of such bacteria, which we can exploit.
I think the long term impact of these studies will depend on how reversible the "state of war" between the immune system and the nerves is.