Earlier quoted context omitted.
There are literally hundreds of papers on BPA and its links with diabetes. The only difference now is that we have the molecular culprit behind the trend. https://www.ucsusa.org/our-work/center-science-and-democracy...
"There are literally hundreds of papers on BPA and its links with diabetes." There are literally not. The review you've linked to cites a handful of papers involving minor effects observed in mouse studies. Moreover, the paper you originally cited proves nothing even approximating a mechanism. I'm not suggesting anything about the safety of BPA, but it's utter nonsense to claim that it is a more plausible explanation…
Elevated blood glucose, as in Type 2 Diabetes, has LONG been known to be primarily driven by excessive production of glucose by the liver due to liver insulin resistance. In fact, the way that the drug metformin improves diabetes is by shutting that pathway down.
http://diabetes.diabetesjournals.org/content/50/8/1807.short
Thats not dietary, thats the hormone insulin being ignored by the liver. This is further complicated by the fact that peripheral tissues, like muscle, are themselves insulin resistant.
The TCF7L2 pathway has been shown to be down-regulated in all of these tissues in diabetics.