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California passes law to ban ultra-processed foods from school lunches

gov.ca.gov

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Re: California passes law to ban ultra-processed foods from school lunches

#261

Earlier quoted context omitted.

Thanks - I'd not seen DASH-sodium but only the DASH study whose samples it's based on. Having read the study now: DASH-sodium barely indicates anything about sodium independently. It looks at three biomarkers which _correlate_ with cardiovascular disease, and finds that reducing sodium from high to low levels for four weeks reduced one of those biomarkers by 19%, _increased_ another biomarker by 9%, and didn't change…

> You could say that this suggests higher sodium increases cardiovascular disease risk but that seems like a stretch when if you'd picked a slightly different set of CVD-correlated biomarkers you would have got exactly the opposite result. What’s the evidence for this? > Even if the results had been more convincing, the methods are not - it's an extremely short term study and looks only at correlative biomarkers and…

> What’s the evidence for this? Exactly what I paraphrased from the study - they chose three biomarkers that correlate with CVD - A increased 19%, B decreased 9%, C stayed the same. If they had chosen some other biomarker D instead of A, that increased say 5% or less, it would give an equally strong but opposite result as the result from the study.

Meta analysis is only as strong as the studies it's based on. I looked at quite a few studies before that purport to show sodium causing CVD, and none of them strongly support their conclusion - they all had significant flaws, not that they're not useful research just that they don't show what they are used to say they show.

For example, there were studies showing that increased salt increased blood pressure by ~5 mm Hg over long term. I understand that blood pressure can be affected very slightly by salt intake, I would guess because the body is holding more water or some other normal mechanism like that, but this does not suggest it's the long term cause of blood pressures going up from a normal 120 to a chronic 160 or 200 as we're seeing in tons of people. There could be any number of adjustments that would increase blood pressure slightly WHILE the change is in effect and then go back to baseline afterward. The chronic high blood pressure is a disease that doesn't just go back to normal immediately after a change.

Anyway, I don't have time at the moment to look through the 11 studies cited in that meta analysis, but if you pick the one or two that give the strongest evidence for salt causing CVD I'd look at them.

I'm genuinely trying to figure this out myself as best I can, because I know way too many people close to me dealing with early stage CVD and diabetes. And a lot of them say they're working on it by avoiding meat and dairy and eggs and salt, and instead of that they end up eating more refined oils and refined flour and sugar. It doesn't seem to be helping them any after years of this, and I think this is backwards advice. I'm not saying we need to eat tons of salt, maybe it does have a minor effect, just that it's not the real culprit.

Re: California passes law to ban ultra-processed foods from school lunches

#262

Earlier quoted context omitted.

I agree with you there, that the healthiness of different foods is complex and a binary category of "ultra processed" is not sufficient for understanding it. In the case of potato chips though, I specifically disagree that they aren't ultra processed - look into the process for creating soybean or canola oil and try to explain how that isn't "ultra processed". further thinking along these lines: Consider a boiled pot…

This is all seed oil panic not based in real science. Olive oil has less omega 3s than canola oil. Omega 3 vs Omega 6 ratio is not a "key mechanism of action" supported by coherent science. It is instead crank stuff extrapolated wildly from research. Frying potato chips in butter won't make them healtheir. People do studies on hexane residue in canola oil. We don't find it in our bodies. "This sounds weird to me" is…

So do you think the whole understanding of oxidative stress, antioxidants, etc is all crank panic vibes? I thought that that was pretty widely accepted. I realize that the idea that some oils oxidize easier than others and have less antioxidants and thus might cause more oxidative stress is often ignored or dismissed outright without being looked at.

And, what is it then that makes potato chips junk food compared to a baked potato with butter? The potato? The oil? The salt? The frying? The fact that the oil was fried in many times over and not fresh? A combination of these - but which specifically?

Re: California passes law to ban ultra-processed foods from school lunches

#263

Earlier quoted context omitted.

> Ok, so we already have your standard Who is "we"? I don't know what you are going on about. Empirical evidence is one thing, mechanism is another source of knowledge by which we can shape public health policy. While empirical evidence is valid only for the situation in which it was obtained, mechanism is universal. For example, we mandate wearing seatbelts in cars in the name of public health. It is however not nec…

> Who is "we"? Me and anyone else reading this. > While empirical evidence is valid only for the situation in which it was obtained, mechanism is universal. Mechanisms are inferred from empirical evidence, I don't see how you can treat them as two separate categories. For example, in your crash test dummy analogy, verification through crash tests (with dummies) is empirical evidence. Yet under your framework, should…

> Me and anyone else reading this.

In that case, no "we" don't, because I am reading this and I do not agree with this "standard" nor your characterization of what I wrote.

> Mechanisms are inferred from empirical evidence, I don't see how you can treat them as two separate categories. For example, in your crash test dummy analogy, verification through crash tests (with dummies) is empirical evidence.

This is not how it works. Crash tests are used for validation, but the data from crash tests is generally not used to infer mechanism. Physicists don't come up with a new theory of mechanics every time a crash test has an unexpected outcome.

> If you name proxy experiments that support your views (crash tests) as mechanisms and ones that don't (SSB replacement with NNS RCTs) as "empirical evidence is valid only for the situation in which it was obtained" then sure, everything you want to believe is supported by sound science and everything you don't isn't.

I don't think you understand. If you want to support a public health intervention you either have the empirical data with a relevant endpoint,

or you can point to mechanism which bridges the part between the data that you have and the outcome which you want to achieve.

When it comes to pharmaceutics and food additives, our mechanistic understanding is insufficient so we often have to resort to empirical studies on humans, including RCTs (Pfizer's Covid vaccine trial had tens of thousands of participants) and also observational studies at population level. And it is the last part where artificial sweeteners fail to show benefit so far.

When it comes to seat belts, our mechanistic understanding is sufficient so we don't need to resort to empirism. Yes we perform validation but only to check if there are no design oversights in the vehicle nor shortcomings with the simulation software, typically in a low triple-digit number of crash tests. But no humans involved and especially no control arm with humans.

(Well if you ignore the one study on the efficacy of parachutes which was done as RCT https://doi.org/10.1136/bmj.k5094 )

> So it doesn't meet your own goalpost

It does, because again, mechanism is provided. You could say that the study has weak evidence for the mechanism and it works like that perhaps only for sugar. Because that some mechanism is found in mice does not mean it is also found in humans, and it would be a fair point. This is why many species are tested and so far the results held up (testing humans takes too long for obvious reasons).

Re: California passes law to ban ultra-processed foods from school lunches

#264

Earlier quoted context omitted.

> You could say that this suggests higher sodium increases cardiovascular disease risk but that seems like a stretch when if you'd picked a slightly different set of CVD-correlated biomarkers you would have got exactly the opposite result. What’s the evidence for this? > Even if the results had been more convincing, the methods are not - it's an extremely short term study and looks only at correlative biomarkers and…

> What’s the evidence for this? Exactly what I paraphrased from the study - they chose three biomarkers that correlate with CVD - A increased 19%, B decreased 9%, C stayed the same. If they had chosen some other biomarker D instead of A, that increased say 5% or less, it would give an equally strong but opposite result as the result from the study. Meta analysis is only as strong as the studies it's based on. I looke…

You said:

> if you'd picked a slightly different set of CVD-correlated biomarkers you would have got exactly the opposite result.

So the evidence I’m looking for is empirics showing CVD correlated biomarkers that suggest a beneficial effect from consuming levels of sodium above recommended levels. Without that evidence then I don’t see why we should believe they would have got the opposite result if they picked other CVD biomarkers.

> but this does not suggest it's the long term cause of blood pressures going up from a normal 120 to a chronic 160 or 200 as we're seeing in tons of people

I’m not claiming that salt is the single cause of hypertension, but that doesn’t mean that the kind of reductions you see from salt reduction aren’t meaningful or contribute to those very high figures. It’s easy to dismiss 5mmHg as insignificant, but we generally see a 5mmHg reduction in sysBP translate to a ~10% reduction in CVD events. Considering how prevalent CVD is, that’s a pretty large effect size.

Chronic diseases are often overdetermined and stack - people have a poor diet which means they consume too much salt, they’re overweight and obesity, have T2DM or prediabetes, sedentary lifestyle, etc etc. The fact that we can’t point to a single one of these and say “this is the thing causing your 160/100 BP doesn’t mean we shouldn’t try to fix the individual factors. So sure, salt reduction seems to offer 5-6mmHg reduction, exercise 4-8, hypertensive drugs 10. But put them all together and that’s a massive change.

> Anyway, I don't have time at the moment to look through the 11 studies cited in that meta analysis, but if you pick the one or two that give the strongest evidence for salt causing CVD I'd look at them.

It’s just three trials (or four depending on how you count TOHP I & II). I think I’ve met my burden in terms of showing there’s evidence of high salt intake having adverse effects, I have no interest in forcing you to read them. Just trying to provide evidence if that’s something you’re seeking.

> I'm genuinely trying to figure this out myself as best I can, because I know way too many people close to me dealing with early stage CVD and diabetes.

I’m sorry to hear that. We certainly seem to be struggling with chronic lifestyle-related disease these days, though with GLP-1 RAs I’m a lot more optimistic than I was a few years ago.

> And a lot of them say they're working on it by avoiding meat and dairy and eggs and salt, and instead of that they end up eating more refined oils and refined flour and sugar.

Yeah depending on the composition of what they’re eating that doesn’t sound great. IMO Replacing butter with refined oils and whole grain flours/carbs - sure, solid move. Replacing meat with plant proteins is also a worthwhile step, and eggs don’t seem to be great for health in many respects. But fermented dairy seems to be a positive as far as CVD risk goes, so ethics aside, seems like a backwards step if they replacing yoghurt and cheese with sugar and white flour!

> I'm not saying we need to eat tons of salt, maybe it does have a minor effect, just that it's not the real culprit.

I’m very wary of trying to find “the culprit” for public health problems. It’s so rarely the case that a disease has a single aetiology, and in my experience the people who’ll tell you “it was the sugar all along”, “it was the seed oils all along”, “it was the glyphosate all along” have a book to sell. The reality is probably closer to it being a combination of several things. Not as sexy though, no publisher or influencer is interested in that view!

Re: California passes law to ban ultra-processed foods from school lunches

#265

Earlier quoted context omitted.

> Who is "we"? Me and anyone else reading this. > While empirical evidence is valid only for the situation in which it was obtained, mechanism is universal. Mechanisms are inferred from empirical evidence, I don't see how you can treat them as two separate categories. For example, in your crash test dummy analogy, verification through crash tests (with dummies) is empirical evidence. Yet under your framework, should…

> Me and anyone else reading this. In that case, no "we" don't, because I am reading this and I do not agree with this "standard" nor your characterization of what I wrote. > Mechanisms are inferred from empirical evidence, I don't see how you can treat them as two separate categories. For example, in your crash test dummy analogy, verification through crash tests (with dummies) is empirical evidence. This is not how…

Sorry, I summarised what I thought was your goalpost earlier in the exact same words and you didn’t correct me, so I made the assumption in subsequent replies. I’m not interested in straw manning your argument, just trying to understand.

I’m going to pass on the crash test dummies bit. You’ve misunderstood the point I was making, but it could be poor communication by me and I think the point is becoming increasingly tangential.

> When it comes to pharmaceutics and food additives, our mechanistic understanding is insufficient so we often have to resort to empirical studies on humans, including RCTs

> It does, because again, mechanism is provided. You could say that the study has weak evidence for the mechanism and it works like that perhaps only for sugar. Because that some mechanism is found in mice does not mean it is also found in humans, and it would be a fair point. This is why many species are tested and so far the results held up (testing humans takes too long for obvious reasons).

So you don’t feel you’re being straw manned again, can I get a clear answer to this: is your argument that if we stack together sufficient numbers of mechanistic animal studies we can be sufficiently confident enough in the translation rate of such studies to humans that we can roll out public health interventions without any evidence of efficacy in human populations?

Re: California passes law to ban ultra-processed foods from school lunches

#266

Earlier quoted context omitted.

Diet is part of the problem but a big part of the issue is the inherent sedentary lifestyle associated with American infrastructure and suburban development. If you design tons of housing in a way that encourages people to utilize cars as much as possible, invest as little as possible into infrastructure like public sidewalks and bike lanes, etc then don’t be shocked when your populace becomes fat and lazy, especiall…

most of the sedentary lifestyle of the US is intentionally done, as a silently understood truth, to avoid violent crime without getting caught by title vii lawsuits the only places that dont need to build suburbs with 10 mile buffer zones from other people are cities like SF and NY that exclude people via rent prices or other place like alaska, obvious reasons i have had (white) frends visit LA/hollywood and get arre…

That’s a lot of words to say you’re scared of black people

Re: California passes law to ban ultra-processed foods from school lunches

#268
post #200

Earlier quoted context omitted.

>> In July 2025, the federal Centers for Disease Control (CDC) reported that 32.7 percent of children and youth between 12 and 19 years old are prediabetic. > Wow. Sadly, I'm not too surprised. My state also has free breakfast and lunch in public schools and it is possible for them to get served over 100g of added sugar between the two meals and classroom provided snacks. Then add to it whatever the kids are eating a…

How much of this is that children refuse to eat normal whole foods? I've heard stories from teachers that mention that children often would rather go without food than eat anything remotely healthy (e.g. apples from cafeteria). My own personal experience from places that are considered food desert is often seeing rotting fruit not being consumed. Doesn't mean we shouldn't try to offer healthy option, but just that th…

“Refusing to eat” is such an absurd phrase. It says more about your impatience than it says about them.

They’re rejecting WHAT you offer WHEN you offer. They’re not refusing to eat. If you simply allow them to say no, they get hungry and come back (usually within an hour). Ask them what they want from the available healthy options and feed them that.

In fact you can set this up ahead of time. “Child what are your five favorite foods? Great two of those are special occasions only, pick two more. Great. That’s your list. Here’s the deal. At meal time you can pick one of those five. I promise to make it, if you promise to eat it.” This gives them a feeling of control and gives you an easy answer.

This actually makes your life easier because you can plan ahead and always have that whitelist available. And it’s less complicated because their pallets are simpler.

There is a whole branch of child psychology devoted to this question. They’re not refusing to eat. They’re simply not interested in the thing you offered.

I was at a friend’s recently. He prepared a lovely meal. Fresh caught salmon and great sides. His daughter didn’t eat a single bite. I was slightly offended. I overheard the fight later “dad, I hate fish, you know that” (actually quite reasonable) his reply was “it’s not a very fishy taste” (a tenuous argument). She was hungry before bed and did eat. The whole mess could have been avoided by not trying to feed her something she hates. I then watched for the rest of the weekend. Every time she was offered something she liked she ate it no problem.

This does of course assume that nobody else is undermining you by offering them junk when they get hungry after refusing a meal.

Re: California passes law to ban ultra-processed foods from school lunches

#269

Earlier quoted context omitted.

This is all seed oil panic not based in real science. Olive oil has less omega 3s than canola oil. Omega 3 vs Omega 6 ratio is not a "key mechanism of action" supported by coherent science. It is instead crank stuff extrapolated wildly from research. Frying potato chips in butter won't make them healtheir. People do studies on hexane residue in canola oil. We don't find it in our bodies. "This sounds weird to me" is…

So do you think the whole understanding of oxidative stress, antioxidants, etc is all crank panic vibes? I thought that that was pretty widely accepted. I realize that the idea that some oils oxidize easier than others and have less antioxidants and thus might cause more oxidative stress is often ignored or dismissed outright without being looked at. And, what is it then that makes potato chips junk food compared to…

I think the extrapolation to "canola oil is poison" is the crank bit.

Re: California passes law to ban ultra-processed foods from school lunches

#270

Earlier quoted context omitted.

Guacamole would count as Group 3 I believe. You use Group 1 and Group 2 ingredients to create it unless you're just grinding up avocados, in which case that's not a guacamole. If you really want to get interesting, you could even say homemade guacamole uses citric acid (from limes) to extend shelf life and enhance the flavor, which is on top of the sodium chloride (salt) that you use to do the same.

> Guacamole would count as Group 3 I believe Too minimally processed-I’d say it’s Group 1. You’re taking Group 1 ingredients (avocado, jalapeño, lime, onion, maybe tomato and cilantro) and chopping, squeezing and pounding them. It can entirely be done with stone tools.

Salt is Group 2, isn't it? (Salt is present unless you make a low sodium guacamole or it's a recipe I've never seen)

Salt is the Group 2 ingredient that I was mentioning. Nova calls out adding salt or sugar to Group 1 makes it Group 3, and specifically calls out Group 1 does not contain added salt.

"Minimally processed foods, that together with unprocessed foods make up NOVA group 1, are unprocessed foods altered by industrial processes such as removal of inedible or unwanted parts, drying, crushing, grinding, fractioning, roasting, boiling, pasteurization, refrigeration, freezing, placing in containers, vacuum packaging or non-alcoholic fermentation. None of these processes add salt, sugar, oils or fats, or other food substances to the original food." https://pmc.ncbi.nlm.nih.gov/articles/PMC10260459/

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