The Case for Transmissible Alzheimer's Grows

Post by doktrin » Mon, Feb 11, 2019, 10:14 AM UTC

rjf72 wrote: post #65

coldcode wrote: post #9

The more we study the human body, the more questions we get. I wonder if some future Dr. McCoy centuries from now will we us as we do medicine of two centuries ago. We can go to other planets and invent new tech every day, but the body still holds a ton of mysteries.

I think we often forget that each generation always thinks they are at the peak of technology except for what they are yet to discover or solve. That is of course completely tautological, but there is a point hidden in there! For instance people in a pre-germ era were not in constant reflection on the inadequacy of e.g. miasma theory. Instead miasma theory was taken as an assumption and so things it failed to explain…

Post by DrScump » Mon, Feb 11, 2019, 11:10 AM UTC

armitron wrote: post #56

itissid wrote: post #40

Earlier quoted context omitted.

1. If certain Prion disease(like say CJD) was Communicable with a high R0[1] by say contact, fecal matter, saliva we would see a lot of people dying, since CJD has a mortality rate of 90% within 1 year[2]. 2. The level of A-beta protein(i.e. not technically prion just yet) again the deposits in 7 of the 8 patients was so severe that if this were communicable easily with a high R0, it would also be seen in the general…

1. CJD is only one specific instance of Prion disease. Others (e.g. vCJD) have incubation periods of - multiple decades -. From the article: "In the original Alzheimer’s transmissibility study, scientists examined the brains of eight patients treated with prion-contaminated human growth hormone as children who decades later died from prion disease" Alzheimers is growing at epidemic proportions, globally [1]. Alzheime…

  Alzheimers in the US is growing at alarming rates

Post by escape_goat » Mon, Feb 11, 2019, 1:01 PM UTC

nonbel wrote: post #107

escape_goat wrote: post #106

Earlier quoted context omitted.

You seem to be mixing terms a bit, so at risk of repeating things you know, I will explain what I am reading from what you've linked. PRNP is the gene encoding for "the major prion protein." Several different errors in PRNP create versions of "the major prion protein" that can be folded in a way that is bad (the prion folding). However, that is not the only way in which the proteins can fold themselves. When lots of…

> "so far all symptoms of prion-type disease for which a cause has been found have turned out to be associated with errors in PRNP." Thanks, this is an interesting new fact to me if true. Is there a source for it? I don't mean a source proving there are no others or something else silly like that. I mean like a review article that mentions they couldn't find any publication about any amyloid beyond PRNP-derived ones…

Post by jayess » Mon, Feb 11, 2019, 4:46 PM UTC

mhkool wrote: post #78

Dr Dale Bredesen has a treatment protocol that improves the conditions of all patients from moderately (late Alzheimer) to significantly (early Alzheimer). His treatment protocol recognizes 3 different types/causes of Alzheimer with an additional 2 subtypes. In December 2018 Dr Bredesen published an article where he shows the results of treatment of 100 patients in various health centers: https://www.omicsonline.org/…

Post by koheripbal » Mon, Feb 11, 2019, 11:39 PM UTC

manmal wrote: post #72

SubiculumCode wrote: post #37

Earlier quoted context omitted.

Or perhaps infection by prions by metal dental equipment.

Unlikely because only eye and neuro tissue would carry prions for contamination of equipment, right? Dental equipment should never come in contact with such tissue.